Infection and AutoimmunityYehuda Shoenfeld, Nancy Agmon-Levin, Noel R. Rose Infection and Autoimmunity encompasses the different mechanisms involved in the infection-autoimmunity association/induction. Special attention is given to heat shock proteins (HSPs) and to transgenic mouse models to better understand infection-induced autoimmunity. Organized into six parts, this book first discusses the mechanisms of autoimmune induction by infection. Some chapters follow discussing the vaccination and vaccines, including the controversial issue of vaccine-autoimmunity relationship. Other chapters elucidate the relationship of bacteria and parasites to autoimmunity. Lastly, the aspects of infections and diseases are described. This reference material will help readers gain a deeper insight into the important etiological aspects of autoimmunity.
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Page 3
... Coxsackievirus B3 [6]. The antigen in this disease has been identified as cardiac myosin, and the autoimmune disease reproduced without virus in susceptible mice by immunization with purified cardiac myosin heavy chain or its ...
... Coxsackievirus B3 [6]. The antigen in this disease has been identified as cardiac myosin, and the autoimmune disease reproduced without virus in susceptible mice by immunization with purified cardiac myosin heavy chain or its ...
Page 4
... Coxsackievirus B-induced myocarditis. Ann NY Acad Sci 1986;475:146-156. Neu N, Rose NR, Beisel KW, Herskowitz A, GurriGlass G, Craig SW. Cardiac myosin induces myocarditis in genetically predisposed mice. J Immunol 1987;139:3630–363 ...
... Coxsackievirus B-induced myocarditis. Ann NY Acad Sci 1986;475:146-156. Neu N, Rose NR, Beisel KW, Herskowitz A, GurriGlass G, Craig SW. Cardiac myosin induces myocarditis in genetically predisposed mice. J Immunol 1987;139:3630–363 ...
Page 31
... coxsackievirus B3 has been shown to be a biphasic disease: an early 'infection' phase and a subsequent autoimmune' phase characterized by T cell and antibody response to cardiac myosin [133]. In TMEV-induced EAE, the induction of ...
... coxsackievirus B3 has been shown to be a biphasic disease: an early 'infection' phase and a subsequent autoimmune' phase characterized by T cell and antibody response to cardiac myosin [133]. In TMEV-induced EAE, the induction of ...
Page 115
... coxsackie virus: initiation by bystander damage and not molecular mimicry. Nat Med 1998;4: 781–785. Horwitz MS, Ilic A, Fine C, Rodriguez E, Sarvetnick N. Presented antigen from damaged pancreatic beta cells activates autoreactive T ...
... coxsackie virus: initiation by bystander damage and not molecular mimicry. Nat Med 1998;4: 781–785. Horwitz MS, Ilic A, Fine C, Rodriguez E, Sarvetnick N. Presented antigen from damaged pancreatic beta cells activates autoreactive T ...
Page 131
... coxsackie B4 virus infection is the extent to which the virus grows in host cells. Interferon alpha and beta production in pancreatic islet beta cells is genetically controlled. Low levels of production predispose to cell mediated ...
... coxsackie B4 virus infection is the extent to which the virus grows in host cells. Interferon alpha and beta production in pancreatic islet beta cells is genetically controlled. Low levels of production predispose to cell mediated ...
Contents
1 | |
5 | |
85 | |
121 | |
Bacteria and Autoimmunity | 319 |
Parasites and Autoimmunity | 437 |
Infection and Diseases | 471 |
Subject Index | 741 |
Other editions - View all
Infection and Autoimmunity Yehuda Shoenfeld,Nancy Agmon-Levin,Noel Richard Rose,Y. Shoenfeld Limited preview - 2004 |
Common terms and phrases
activation acute agents animals anti antibodies antigens arthritis Arthritis Rheum associated autoantibodies autoimmune disease bacterial binding blood cause cells chronic Clin clinical clones common complex controls cross-reactive cytokines damage demonstrated detected determined diabetes effects epitope et al evidence experimental expression factor frequency function gene genetic heart hepatitis host human immune response Immunol important increased individuals induced infection inflammatory initial involved lead levels liver lymphocytes major mechanisms mice molecular mimicry molecules multiple sclerosis myocarditis myosin Nature normal observed occur pathogenesis patients peptide peripheral persistent possible present production protein pylori reactive recent receptor recognized reported rheumatic fever risk role sequence serum severe showed shown similar specific spreading stimulation suggested syndrome synovial fluid T-cell therapy thyroid tion tissue treatment trigger type 1 diabetes vaccine vasculitis viral viral infection virus viruses
Popular passages
Page 147 - Navarro for their help in preparing the manuscript. This work was supported in part by CONACyT, Mexico and Fundacion RJ Zevada.
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Page 287 - Family study of the major histocompatibility complex in patients with systemic lupus erythematosus: importance of null alleles of C4A and C4B in determining disease susceptibility.
Page 147 - Immunologic tolerance maintained by CD25+ CD4+ regulatory T cells: their common role in controlling autoimmunity, tumor immunity, and transplantation tolerance. Immunol Rev 2001;182:18-32.
Page 283 - Genome scan of human systemic lupus erythematosus: evidence for linkage on chromosome Iq in African-American pedigrees. Proc Natl Acad Sci USA 1998:95(25): 14869-14874.
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Page 607 - A randomized trial comparing intravenous immune globulin and plasma exchange in Guillain-Barre syndrome.
Page 529 - Jerne NK, Towards a network theory of the immune system.
Page 245 - BURGESS, JA : High frequency of diabetes mellitus in young adults with congenital rubella.